The Pathophysiology and Clinical Aspects of Hypercalcemic Disorders
Identifieur interne : 000D50 ( Main/Exploration ); précédent : 000D49; suivant : 000D51The Pathophysiology and Clinical Aspects of Hypercalcemic Disorders
Auteurs : David B. N. Lee ; Edward T. Zawada ; Charles R. KleemanSource :
- Western Journal of Medicine [ 0093-0415 ] ; 1978-10.
English descriptors
- Teeft :
- Abnormality, Acromegaly, Acta, Adenoma, Adenomatosis, Alkalosis, Arch intern, Associ, Asymptomatic, Body weight, Bone resorption, Breast cancer, Brit, Calcemia, Calcitonin, Calcium, Calcium homeostasis, Calcium metabolism, Caprot, Carcinoma, Cation, Cemia, Citrate, Cium, Clin, Clin endocr, Clin endocr metab, Coburn, Corticosteroid, Cortisone, Cyclic, Diuretic, Dos, Ectopic, Endocr, Endocrinology, Engl, Excretion, Furosemide, Glucocorticoid, Gordan, Hemodialysis, Homeostasis, Hyper, Hypercal, Hypercalcemia, Hypercalcemic, Hypercalcemic disorders, Hypercalciuria, Hyperpara, Hyperparathyroid, Hyperparathyroidism, Hyperplasia, Hyperthyroidism, Hypervitaminosis, Hypocalcemia, Hypoparathyroidism, Idiopathic, Immobilization, Infusion, Insufficiency, Intern, Intestinal, Intoxication, Intravenous, Jama, Lancet, Lithium, Magnesium, Malignancy, Malignant, Massry, Medullary, Ment, Metab, Metabolism, Metastasis, Metastatic, Mithramycin, Myeloma, Neoplastic, Nonparathyroid, Normal subjects, October, Other hand, Para, Parathyroid, Parathyroid adenoma, Parathyroid glands, Parathyroid hormone, Parathyroidectomy, Parathyroidism, Pediatrics, Percalcemia, Pheochromocytoma, Phos, Phosphorus, Phpt, Primary hyperparathyroidism, Proc, Prostaglandin, Raisz, Reabsorption, Renal, Renal failure, Resorption, Sarcoid, Sarcoidosis, Scand, Secondary hyperparathyroidism, Serum calcium, Serum calcium concentration, Stenosis, Sulfate, Surg, Surgical, Syndrome, Thiazide, Thyroidism, Thyrotoxicosis, Tients, Tion, Toxicity, Transplantation, Ultrafiltrable, Urinary, Urinary excretion, Venous, Vitamin, Western journal.
Abstract
For the purposes of this review, the vast and increasingly complex subject of hypercalcemic disorders can be broken down into the following categories: (1) Physiochemical state of calcium in circulation. (2) Pathophysiological basis of hypercalcemia. (3) Causes of hypercalcemia encountered in clinical practice: causes indicated by experience at the University of California, Los Angeles; neoplasia; hyperparathyroidism; nonparathyroid endocrinopathies; pharmacological agents; possible increased sensitivity to vitamin D; miscellaneous causes. (4) Clinical manifestations and diagnostic considerations of hypercalcemic disorders. (5) The management of hypercalcemic disorders: general measures; measures for lowering serum calcium concentration; measures for correcting primary causes—the management of asymptomatic hyperparathyroidism.
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Kleeman</name><affiliation><wicri:noCountry code="subField">Angeles).</wicri:noCountry></affiliation></author></analytic><monogr></monogr><series><title level="j">Western Journal of Medicine</title><title level="j" type="abbrev">West J Med</title><idno type="ISSN">0093-0415</idno><imprint><publisher></publisher><date type="published" when="1978-10">1978-10</date><biblScope unit="volume">129</biblScope><biblScope unit="issue">4</biblScope><biblScope unit="page" from="278">278</biblScope><biblScope unit="page" to="320">320</biblScope></imprint><idno type="ISSN">0093-0415</idno></series></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0093-0415</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="Teeft" xml:lang="en"><term>Abnormality</term><term>Acromegaly</term><term>Acta</term><term>Adenoma</term><term>Adenomatosis</term><term>Alkalosis</term><term>Arch intern</term><term>Associ</term><term>Asymptomatic</term><term>Body weight</term><term>Bone resorption</term><term>Breast cancer</term><term>Brit</term><term>Calcemia</term><term>Calcitonin</term><term>Calcium</term><term>Calcium homeostasis</term><term>Calcium metabolism</term><term>Caprot</term><term>Carcinoma</term><term>Cation</term><term>Cemia</term><term>Citrate</term><term>Cium</term><term>Clin</term><term>Clin endocr</term><term>Clin endocr metab</term><term>Coburn</term><term>Corticosteroid</term><term>Cortisone</term><term>Cyclic</term><term>Diuretic</term><term>Dos</term><term>Ectopic</term><term>Endocr</term><term>Endocrinology</term><term>Engl</term><term>Excretion</term><term>Furosemide</term><term>Glucocorticoid</term><term>Gordan</term><term>Hemodialysis</term><term>Homeostasis</term><term>Hyper</term><term>Hypercal</term><term>Hypercalcemia</term><term>Hypercalcemic</term><term>Hypercalcemic disorders</term><term>Hypercalciuria</term><term>Hyperpara</term><term>Hyperparathyroid</term><term>Hyperparathyroidism</term><term>Hyperplasia</term><term>Hyperthyroidism</term><term>Hypervitaminosis</term><term>Hypocalcemia</term><term>Hypoparathyroidism</term><term>Idiopathic</term><term>Immobilization</term><term>Infusion</term><term>Insufficiency</term><term>Intern</term><term>Intestinal</term><term>Intoxication</term><term>Intravenous</term><term>Jama</term><term>Lancet</term><term>Lithium</term><term>Magnesium</term><term>Malignancy</term><term>Malignant</term><term>Massry</term><term>Medullary</term><term>Ment</term><term>Metab</term><term>Metabolism</term><term>Metastasis</term><term>Metastatic</term><term>Mithramycin</term><term>Myeloma</term><term>Neoplastic</term><term>Nonparathyroid</term><term>Normal subjects</term><term>October</term><term>Other hand</term><term>Para</term><term>Parathyroid</term><term>Parathyroid adenoma</term><term>Parathyroid glands</term><term>Parathyroid hormone</term><term>Parathyroidectomy</term><term>Parathyroidism</term><term>Pediatrics</term><term>Percalcemia</term><term>Pheochromocytoma</term><term>Phos</term><term>Phosphorus</term><term>Phpt</term><term>Primary hyperparathyroidism</term><term>Proc</term><term>Prostaglandin</term><term>Raisz</term><term>Reabsorption</term><term>Renal</term><term>Renal failure</term><term>Resorption</term><term>Sarcoid</term><term>Sarcoidosis</term><term>Scand</term><term>Secondary hyperparathyroidism</term><term>Serum calcium</term><term>Serum calcium concentration</term><term>Stenosis</term><term>Sulfate</term><term>Surg</term><term>Surgical</term><term>Syndrome</term><term>Thiazide</term><term>Thyroidism</term><term>Thyrotoxicosis</term><term>Tients</term><term>Tion</term><term>Toxicity</term><term>Transplantation</term><term>Ultrafiltrable</term><term>Urinary</term><term>Urinary excretion</term><term>Venous</term><term>Vitamin</term><term>Western journal</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract">For the purposes of this review, the vast and increasingly complex subject of hypercalcemic disorders can be broken down into the following categories: (1) Physiochemical state of calcium in circulation. (2) Pathophysiological basis of hypercalcemia. (3) Causes of hypercalcemia encountered in clinical practice: causes indicated by experience at the University of California, Los Angeles; neoplasia; hyperparathyroidism; nonparathyroid endocrinopathies; pharmacological agents; possible increased sensitivity to vitamin D; miscellaneous causes. (4) Clinical manifestations and diagnostic considerations of hypercalcemic disorders. (5) The management of hypercalcemic disorders: general measures; measures for lowering serum calcium concentration; measures for correcting primary causes—the management of asymptomatic hyperparathyroidism.</div></front></TEI><affiliations><list></list><tree><noCountry><name sortKey="Kleeman, Charles R" sort="Kleeman, Charles R" uniqKey="Kleeman C" first="Charles R." last="Kleeman">Charles R. Kleeman</name><name sortKey="Lee, David B N" sort="Lee, David B N" uniqKey="Lee D" first="David B. N." last="Lee">David B. N. Lee</name><name sortKey="Zawada, Edward T" sort="Zawada, Edward T" uniqKey="Zawada E" first="Edward T." last="Zawada">Edward T. Zawada</name></noCountry></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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